تدريب Shadowing: Other Corneal Disorders - CRASH! Medical Review Series - تعلم التحدث بالإنجليزية عبر الفيديو

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we'll talk here about some other corneal problems

so we'll talk about the wide range of corneal dystrophies as well as keratotorus

and keratoconus so the cornea is seen here it's right here it's this transparent portion consistent with the sclera

that covers the anterior chamber it's important that the sclera is transparent

because it allows light to go through the lens

and ultimately to the retina that being said even though the cornea

and the sclera are consistent the cornea is much different tissue than the sclera the cornea has to be transparent

and so it's made up of very organized collagen fibers as well as cells.

The cornea gets its nutrients through the anterior aqueous,

not through vessels, and that allows it to stay clear in its non -pathologic state.

Okay, so corneal dystrophies, I don't want you to memorize all of these by any means

because you don't have to memorize these, you don't have to memorize the treatment, you just need to know when to suspect corneal dystrophy because these are going to be treated by an ophthalmologist.

So these are a group of progressive congenital eye disorders in which

abnormal material accumulates in one or several levels of the cornea

and this disrupts the transparent property of the cornea as well as the corneal structure itself

so essentially what's going on is

because you have a genetic mutation the cells are creating something

that they shouldn't be creating and because of that you're disrupting

that fine organized corneal layer the vast majority of corneal dystrophies are congenital

and among those most of them are going to be autosomal dominant

and that's useful because you have a patient who you're suspecting some kind of corneal problem in

because they have corneal haze corneal opacities one of the best

questions you can ask is do your parents have any problems

do your parents have blindness does your parent has have your parents ever had anything like this

that will be really helpful in establishing this diagnosis.

They tend to affect both eyes simultaneously, but they don't have to, but that usually is how it goes.

Common symptoms to most corneal dystrophies, including a long -standing foreign body sensation or mild pain in the affected eye or eyes,

and that just has to do with the chronic nature of this.

The foreign body sensation is due to the fact

that the cornea is a very sensitive structure

if you don't believe me try and touch the middle of your eye

and see how long you can do it it's very very highly innervated

and very sensitive

so even the smallest changes these dystrophies will cause foreign body sensation the patient may experience halos

when looking into light and that's just because of the refraction from the foreign body, from the compound that's in the cornea,

refracting back to the retina.

Also, there's reduced visual acuity, and you'll see this particularly in patients where there's significant corneal haze.

And then otherwise, the patient is normal, so their pupils will react to light and to accommodation.

Extraocular movements should be normal.

The only thing that's abnormal is the fact that these patients generally have some level of corneal haze, difficulty seeing, and a foreign body sensation.

And this is a chronic problem.

So this isn't something that came up a week ago.

This is something that the patient has been dealing with for several months.

So among corneal dystrophies, and there's about 17 or 18 that are known.

There's probably even more.

The similarities are that they tend to be congenital.

The symptoms are consistent.

There's a long -standing foreign body sensation or mild pain in the eyes.

Decreased acuity as these corneal opacities come in.

They don't affect other parts of the body.

These are syndromes, but they don't affect anywhere else in the body except for

the eyes they also tend to affect both eyes similarly at the same time

and they're not caused by external forces this is a genetic problem this has nothing to do with trauma or infection

The differences lie in the particular location of the cornea that's affected.

Some tend to be more superficial, some tend to affect the stroma more, and some tend to affect the endothelium more.

Some have symptoms very, very early on, and some don't.

The age of presentation can be different.

Some of these corneal dystrophies present in childhood, others present in later adulthood could.

The extraneous materials that you would see if you were to do a histology would be different.

So some of them will make lipids, some will make cholesterol, and that's certainly not the end.

There's many more extraneous materials that could be secreted into the cornea.

The prognosis can be different based on whether

or not we have a good treatment modality for that particular corneal dystrophy and that kind of works into the treatment.

So here's our cornea here.

This is just cut right down the middle.

This is the outside here and then this is the inside here bordering on the anterior aqueous chamber.

So you have your stratified squamous epithelium here.

There's several layers.

These cells regenerate and they move upward.

So these outer cells are flat like a pancake they're squamous

cells you see a lot of nucleus material the more inner cells are more of a columnar shape beneath

that you have Bowman's membrane

which serves as basement membrane for the epithelium the largest section of the cornea is made up of collagen

and it is the stroma

and then down here you see a little layer of cells this is endothelium

and But in between the endothelium and the stroma, you have what's known as decimates membrane.

So the anterior corneal dystrophies affect the epithelium and the Bauman's layer.

There's four.

There's the epithelial basement membrane dystrophy, which is autosomal dominant, but it can be de novo.

So the patient wouldn't have any parents who had it.

The patient just developed the mutation on their own.

Lisch corneal dystrophy, which stands alone as the one X -linked dominant corneal dystrophy, Rice -Buckler corneal dystrophy, and Theobanky corneal dystrophy.

Those are both autosomal dominant.

When it comes down to the stroma, there are several different types.

Most, all but two, are autosomal dominant.

Gelatinous droplet corneal dystrophy, there's the two granular corneal dystrophies, type one and two the two lattice corneal dystrophies type one

and two macular corneal dystrophy which is autosomal recessive and schneider crystalline corneal dystrophy which is autosomal dominant

like i said you don't need to memorize these

and then the posterior corneal dystrophies these affect the endothelium

and the decimates membrane there's congenital hereditary endothelial dystrophy

or ched type one that's an autosomal dominant variant of the same disease as chad type 2 which is autosomal recessive.

And then Fuchs endothelial dystrophy is an autosomal dominant disorder.

This has a distinguishing factor in that this tends to affect females more than men.

It also tends to affect older women more than younger women.

And then finally posterior polymorphous dystrophy which is autosomal dominant.

So here's all your corneal dystrophies, the major types of corneal dystrophies, do not memorize these.

Here's some distinguishing factors though.

So lattice corneal dystrophy type 1, macular corneal dystrophy, and Schneider crystalline corneal dystrophy,

as well as both of the CHED dystrophies, these all have a tendency to affect young people.

100

So CHED 1 and 2 will be apparent at birth and

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so by apparent I mean corneal opacities

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and in case you're wondering how do you tell a corneal

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opacity from a lenticular opacity the corneal opacity will appear over

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even the iris whereas the lenticular opacity you won't see any opacities over the iris just over the pupil.

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So CHED 1 and 2 will be apparent at birth.

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Signs of lattice corneal dystrophy may be present at birth, but symptoms do not typically present until the child is older,

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post -pubescent, or even early adulthood.

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Schneider crystalline corneal dystrophy can be a present in the first decade of life, so young children again here,

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and then signs of macular corneal dystrophy can be present around puberty.

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So these are some of the corneal dystrophies that tend to show up in younger people.

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On the other hand, we have Fuchs endothelial dystrophy.

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These tend to present in older people, in people who are in their 50s, 60s, 70s and beyond.

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This also has a tendency to present in women more than men, about 4 to 1 ratio.

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So a Fuchs endothelial dystrophy would be notorious for striking the

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otherwise healthy 60 to 70 year old lady with no other health problems other than a progressive decreasing visual acuity,

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which she ascribes to aging.

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Very important to look in the patient's eyes.

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Don't always say, oh, you're an old person.

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It must be presbyacusis.

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Maybe your lens is getting stiff.

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Look into the eyes.

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You know corneal precipitates.

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Obviously, those shouldn't be there.

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And then you look back on her history questionnaire.

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And indeed, her father was totally blind by age 75.

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And this is an autosomal dominant disorder, meaning that if a parent has it, you have a 50 -50 chance of getting it.

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So all suspected corneal dystrophies are going to require consult to an ophthalmologist for specialized diagnostics and treatment.

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If they're a younger person, especially if it's an autosomal dominant disease, you should provide them with genetic counseling at some point in the future

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and occupational therapy can be useful for helping these people maximize their potential.

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Your job as the clinician, as the internist, as the pediatrician,

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as the ED doctor, is not to make the official diagnosis of which type of corneal dystrophy the patient has.

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That's done by the ophthalmologist.

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Your job is to suggest

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that this patient may have a corneal dystrophy to rule out

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any other possible causes of corneal haze such as keratitis

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and then to identify these patients and send them off to the ophthalmologist.

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There's very little you can do for corneal dystrophies in a clinical setting and the condition itself is not an emergency.

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So this might be a patient that

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if they say oh I've only noticed it for one or two days you might get a fluorescein stain on.

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But if they say they've had it for several weeks, maybe not.

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It's probably going to be a corneal dystrophy.

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Okay, keratotorus, this is also known as pellucid marginal degeneration,

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is an idiopathic degenerative corneal condition characterized by abnormal thinning of the stroma.

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And the stroma is part of the cornea.

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So when the stroma thins, the corneus thins.

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You also have a thinning of Baumann's layer and possibly other structures, but it's primarily the stroma.

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And the bulk of this thinning happens below the horizontal midline of the cornea and then out along the periphery.

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So if you think of a clock, you would think of

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if you were to take out the inner circle of the

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clock from about four o 'clock to eight o 'clock

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so the the periphery on the bottom of the eye

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and this shape creates an irregular astigmatism remember astigmatism is just due to an abnormally shaped lens

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or an abnormally shaped cornea and the result of that is

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that you're projecting different dimensions in different areas either in front on

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or behind the retina and so this creates an irregular astigmatism so a patient presenting with new astigmatism, this is a possible diagnosis.

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Typically, carototaurus affects both eyes, not just one.

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The symptoms include gradual decrease in visual acuity, distorted images, symptoms that are consistent with astigmatism.

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Oftentimes, the patient has seen an optometrist for glasses, but the prescription keeps changing because carototaurus,

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pellucid marginal degeneration, is a progressive disease.

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In some cases the patient will have extreme pain and those cases it will reflect a corneal perforation but that's rare.

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So for diagnosis what you're going to do is a slit lamp examination

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and you're going to do corneal topography

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and the corneal topography will help you identify how thick the cornea is

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and in which areas and so if you notice that the cornea is thinner, down at the bottom, along that four o 'clock to eight o 'clock line,

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then your suspicion for cratitoris goes up.

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The treatment, 88 % of these patients will be managed by eyeglasses or contact lenses.

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They'll need frequent follow -up with an optometrist

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because they're going to need changes in their prescription relatively frequently

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or more frequently than most of us 12 % of these patients will be candidates for phototherapeutic keratectomy,

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but the primary treatment for keratotaurus is eyeglasses or contact lenses.

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Keratoconus is an idiopathic degenerative disorder of the cornea characterized by degeneration of the peripheral corneal structures.

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So if you think of the cornea as a globe,

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as a dome if you're degenerating all the area around it with the exception of the middle then

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that ball that globe is going to turn into a pyramid

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and this is where we get the word keratoconus so so

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that change in shape will certainly cause refractory issues remember

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that the cornea has a refractory index of about 40 45 to 50 diopters

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so as the shape of the cornea changes it's going to

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cause refractory issues can cause astigmatism as well as monocular diplopia most commonly keratoconus is diagnosed in the adolescent

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years.

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So the symptoms are distortion of images, decreased visual acuity at all distances,

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monocular diplopia, difficulty with night vision, and halos.

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And again here we're going to do a slit lamp examination for corneal topography.

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We want to get a good idea of what

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that cornea looks like because a coratotaurus versus a coratoconus versus a normal cornea look completely different.

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And the treatment here, there's abundant options.

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Contact lenses can be used, special contact lenses, to scleral lenses, to several different types of surgery, including also corneal transplant.

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So here's an example of keratoconus.

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If you look up really closely, you can see that this cornea is relatively normal on the front,

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but it's thinned out along the sides.

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and it makes that cone shape.

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It's not a perfect cone, but you can see where they get the word keratoconus.

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المفردات وملاحظات التحدث لهذا الدرس

يحتوي هذا الفيديو على 192 جملة و2365 كلمة للتدرب على الترديد. مدة الكلام 17:06. يتكلم المتحدث بسرعة ثابتة، نحو 138 كلمة في الدقيقة، وهي سرعة مريحة للترديد. 75% فقط من الكلمات ضمن أكثر 3,000 كلمة شيوعًا في الإنجليزية، فالمفردات صعبة نسبيًا.

أهم المفردات في هذا الفيديو

15 كلمة أقل شيوعًا من الفيديو، مع النطق والمعنى:

dominant /ˈdɑmənənt/ (صفة) — سَائِد. Ruling; governing; prevailing
lens /lɛnz/ (اسم) — عَدَسَة. An object, usually made of glass, that focuses or defocuses the light that passes through it.
symptom /ˈsɪm(p)təm/ (اسم) — عَرَض, عَلَامَة. A perceived change in some function, sensation or appearance of a person that indicates a disease or disorder, such as fever, headache or rash; strictly, a…
thin /ˈθɪn/ (صفة) — رَفِيع, رَقِيق. Having little thickness or extent from one surface to its opposite.
layer /ˈleɪ̯ɚ/ (اسم) — طَبَقَة. A single thickness of some material covering a surface.
sensation /sɛnˈseɪʃən/ (اسم) — حَاسَّة, حِسّ. A physical feeling or perception from something that comes into contact with the body; something sensed.
membrane /ˈmɛm.bɹeɪn/ (اسم) — غِشَاء. A mechanical, thin, flat flexible part that can deform or vibrate when excited by an external force.
consistent /kənˈsɪs.tənt/ (صفة) — مُتَّسِق. Of a regularly occurring, dependable nature.
disorder /dɪsˈɔɹdɚ/ (اسم) — اِخْتِلال, اِضْطِراب. Absence of order; state of not being arranged in an orderly manner.
diagnosis /daɪəɡˈnəʊsɪs/ (اسم) — تَشْخِيص. The process of, or an instance of, identification of the nature and cause of a medical condition or illness.
transparent /tɹænsˈpæɹənt/ (صفة) — شَفَّاف. See-through, clear; having the property that light passes through it almost undisturbed, such that one can see through it clearly.
anterior /ænˈtɪɹ.i.ɚ/ (صفة) — أَمَامِيّ. Nearer the forward end, especially in the front of the body; nearer the head or forepart of an animal.
progressive /pɹəˈɡɹɛsɪv/ (اسم) — تَقَدُّمِيّ, مُتَرَقٍّ. One with liberal or progressive political beliefs.
abnormal /ˌæbˈnɔɹ.ml̩/ (صفة) — غَيْر طَبِيعِيّ. Not conforming to rule or system; deviating from the usual or normal type.
chamber /ˈt͡ʃeɪ̯mbɚ/ (اسم) — حُجْرَة. The private room of an individual, especially of someone wealthy or noble.

الأفعال المركبة التي ستسمعها

come up (فعل) — طَلَعَ. To emerge or become known, especially unexpectedly.
go up (فعل) — صَعِدَ, يَصْعَدُ. To move upwards.
take out (فعل) — أَخْرَجَ. To remove.
talk about (فعل) — تَكَلَّمَ فِي, تَكَلَّمَ عَلَى. Used to draw attention to the speaker's characterization of someone or something.

نطق يحتاج إلى انتباه

يستخدم المتحدث 34 من الصيغ المختصرة، مثل don't, you're, they're. انطقها بصيغتها القصيرة كما تسمعها.

أصوات “th”: thin /ˈθɪn/, thick /θɪk/, beneath /bɪˈniːθ/, ophthalmologist /ˌɔf.θə(l)ˈmɑ.lə.d͡ʒɪst/, epithelium /ˌɛpɪˈθiːlɪəm/
أصوات “sh” و“zh”: sensation /sɛnˈseɪʃən/, examination /ɪɡˌzæm.ɪˈneɪ.ʃən/, prescription /pɹəˈskɹɪp.ʃən/, distinguish /dɪˈstɪŋ.ɡwɪʃ/, mutation /mjuˈteɪʃən/
الكلمات الطويلة — انتبه لموضع النبر: anterior /ænˈtɪɹ.i.ɚ/, difficulty /ˈdɪfɪkəlti/, examination /ɪɡˌzæm.ɪˈneɪ.ʃən/, irregular /ɪˈɹɛɡ.jə.lɚ/, primarily /ˈpɹaɪ.mə.ɹə.li/

كيف تتدرب بهذا الفيديو

استمع إلى الفيديو كاملًا مرة واحدة دون أن تتكلم، ودوّن الكلمات التي لا تعرفها.
ردّد جملة جملة بالسرعة العادية، وكرّر كل جملة حتى يتطابق إيقاعك مع المتحدث.
سجّل صوتك وقارنه بالأصل، مع الانتباه إلى كلمات مثل dominant, lens, symptom.

القواعد في هذا الفيديو

أكثر التراكيب التي يستخدمها المتحدث، مع العبارات نفسها من الفيديو:

التركيب	في الفيديو
المبني للمجهول be + اسم المفعول — التركيز على ما يحدث لا على من يفعله	is seen · being said · be treated
جمل الوصل who / which + جملة — معلومة إضافية عن شخص أو شيء	dystrophy, which is · dystrophy which is · people who are
المضارع التام have/has + اسم المفعول — حدث ماضٍ ما زال له أثر الآن	I've only noticed · they've had · has seen

ما هي تقنية التظليل الصوتي؟

التظليل الصوتي (Shadowing) تقنية تعلم لغة مدعومة علمياً، طُورت أصلاً لتدريب المترجمين الفوريين المحترفين. الطريقة بسيطة لكنها قوية: تستمع لصوت إنجليزي أصلي وتكرره فوراً بصوت عالٍ — كظل يتبع المتحدث بتأخير 1-2 ثانية. تُظهر الأبحاث تحسناً كبيراً في دقة النطق والتنغيم والإيقاع وربط الأصوات والاستماع والطلاقة.

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تدريب Shadowing: Other Corneal Disorders - CRASH! Medical Review Series - تعلم التحدث بالإنجليزية عبر الفيديو

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المفردات وملاحظات التحدث لهذا الدرس

أهم المفردات في هذا الفيديو

الأفعال المركبة التي ستسمعها

نطق يحتاج إلى انتباه

كيف تتدرب بهذا الفيديو

القواعد في هذا الفيديو

ما هي تقنية التظليل الصوتي؟