शैडोइंग अभ्यास: Lecture 12: Retina, Part 2: Retinal vascular occlusions - वीडियो के साथ अंग्रेजी बोलना सीखें

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I'm going to show you what I'm going to do with you in the second part of our program.

It's one of the most important things that we have in the basic knowledge course.

I'm going to show you what I'm going to do with you.

If you're listening to the first one, you'll be listening to it.

The first one is really important.

It's the introduction.

We're going to show you what we're going to talk about.

The first thing is that the main artery is that the main artery is occluded.

What can it be in the artery?

This is the most common cause.

The artery is a lot of different from it.

which can become thrombus in the vein, and can become something in the Aorta,

Aortic Coarchitation, Aortic Thrombus, or aortic disease,

diseases in the internal jugular and the things that they can get

and go to the central retinal artery

and get slowed in a lot of times there is a thing called There is something called Fyprenolytic Enzymes,

which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes,

which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes,

which is called Fyprenolytic Enzymes, which is called Fyprenolytic Enzymes, which pressure, for example, the Orbitacillulitis,

or Retrobulbar hemorrhage, if it's too low, it can also be basculitis.

This is the common etiologist, but it's of course, the Impulite.

So, what do you think?

What do you think?

This is the center of the artery, which is the center of the artery, which is suddenly occluded with the eye.

What do you think?

As you can see, sudden loss of vision.

Suddenly.

When we talk about sudden loss of vision, we have to know the reasons.

The reason for sudden loss of vision is 4 things.

Central retinal arthroclusion.

Amorosis phygex.

What is this amorosis phygex?

This is acute or recurrent ischemic attacks of loss of vision.

It is important, but the important thing is to resolve,

so you can do attacks of loss of vision and then you can do it with central artery occlusion.

Amorosis phygox is something, central artery occlusion is something else.

The third thing is trauma with lesion of optic nerve or transaction of optic nerve.

This is the 4th thing, which is the 4th thing, which is the 4th thing,

which is the 4th thing, which is the 4th thing, which is the 4th thing, which is the 4th thing, which is the 4th thing,

which is the 4th thing, which is the 4th thing, which is the 4th thing, which is the 4th thing, which is the 4th thing.

If you have any questions, you can answer your question, and you can answer your question, and you can answer your question,

and you can answer your question, and you can answer your question, and you can answer your question, and you can answer your question,

and Central Island Division Of course, we all have to say that

The temporal island division

Is it who is?

It's the one who has it

And I can't say it Tubular vision

No, it's not constructional vision

Because this island

It's not a form of tube

It's not a central vision

It's a temporal vision

It's a temporal area

This is for example.

For example signs, the first thing is related referring to pillar defect which is R .A .B .D.

Related to pillar defect related to pillar defect which is So the afferent doesn't work well,

the afferent doesn't work well, but the afferent doesn't work well.

This means that the brain will react consensually, but not directly.

How is it?

The afferent will happen with any type of afferent.

Like the optic neuropathy or the optic atrophy.

Like the retinal detachment and central retinal arthroclusion.

That's why you are able to get the impulse to the brain from the brain

When you put light on the eye that is affected, you can find it

What is it?

It's not a construction or a construction

It's a construction

When you put light on the eye

It's a construction

When you put light on the eye that is affected

When you put light on the eye

It's a dilate because from the contentual constriction which is strong

to the direct constriction which is weak so you can see

that it is not dilate this is the relative effect of the effect of the effect of the market

and the way that I detect the market is called swinging flash light reflex

when you put light on your eyes

and then you put it on your eyes again

and then you put it on your eyes again and then you put it on your eyes again

So what do we call?

Relative Aferent Propeller Defecta Or we call it Marcos Gun Bupilum This is a sign that is very important

For example, this is the malingering

From the actual Lesion or brain occlusion

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The sign that we call Retinal Whitening and Placification You can see the retina

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You can see it

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It's not bad, it's not bad and this is why this is

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because of the redema which will happen in the center of the urtocleus the redema ischemia

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and swelling and edema so the edema and swelling of the fetishus is a little bit on the .

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Fobia is not supplied by the retina,

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because it's not supplied by the retina, because it's not supplied by the retina, because it's not supplied by the retina,

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because it's not supplied by the retina, because it's not supplied by the retina, because it's not supplied by the retina,

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because it's not supplied by the retina, because it's and it becomes affected by the rhythm and whitening and low -passification.

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But the phobia is not working.

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Why?

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Because the connection of the phobia, the nerve fibers and the ganglion cells are in the macula, which is affected.

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The nerve fiber layer which is working and is affected.

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So that's why it is not working.

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But the pan clinical in the rhythm is that it is

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so we can put

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it in the middle of the heart of the heart of

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the heart of the heart of the heart of the heart

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of the heart of the heart of the heart of the

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